Inflammation’s hidden retinal imprint
Another important finding from the study points to genetic variants linked to inflammatory processes in the brain, which may also play a role in structural changes observed in the retina.

These results lend additional support to the inflammation hypothesis of schizophrenia — the idea that inflammation could drive the onset or progression of the disorder.

“If this hypothesis is confirmed, inflammation could be interrupted by medication, potentially enabling us to improve treatment possibilities in the future,” Rabe explained.